Tag Archives: Alzheimer’s
In “The Origin of Disease: The War Within” we wrote that abnormal proteins generated by immortal pathogens are not alive; and thus, cannot be killed. Abnormal proteins cause tissue to stick together, become inclusion cysts, are a focus for immune system attack, and adhere to and alter genes and gene expression. Understanding immortal pathogens are generating abnormal proteins may lead to alternatives for treatment, and new ways to eliminate abnormal proteins in the body after acute and chronic infection. …..
Check out the interview of Nikki Schultek, founder of of the Intracell Research Group, on infection and Alzheimer’s disease. Their website shares scientific articles on chronic infection as a cause of chronic disease, and four of their board members were cited in our book. We added a link to their webpage on our FB page because of their important work and advocacy; and their readily available resources supporting infectious causes of chronic disease. https://www.youtube.com/watch?feature=sshare&v=W3ICLHkykDg&app=desktop
Chlamydia species and H-pylori have been linked to ocular surface disorders, glaucoma, and retinal disease. Chlamydia trachoma is the leading cause of blindness in the world. Chlamydia psittacosis can cause MALT lymphoma in the eye, Waldenstrom syndrome, and ocular melanoma. Toxoplasmosis has been linked to retinal disease and blinding. Many eye diseases are described as “itis”, meaning inflammation in a specific part of the eye, without understanding pathogens are capable of causing the inflammation and the disease or syndrome (more…..
Cerebral cavernous malformation (CCM) is a cluster of malformed and tangled blood vessels in the brain, which was previously assumed to be genetic. Patients with CCM can be asymptomatic; or can have long-term health problems, including headaches, epilepsy, stroke, and death. In 2017, a multistate study, in mice, found gram-negative bacteria activated TLR-4; which was the molecular mechanism in endothelial cells that underlies formation of CCM. CCM and Alzheimer’s have similar findings, in the brain—abnormal and tangled blood vessels, neuronal…..
The scientific evidence is mounting—chlamydia species cause neurodegenerative diseases. Chlamydia pneumonia has repeatedly been linked to Alzheimer’s disease; and chlamydia pneumonia is known to generate beta-amyloid plaque, such as found in Alzheimer’s brains. Chlamydia trachoma has been linked to Parkinson’s disease; and chlamydia trachoma generates excess alpha-synuclein proteins, which is found in Parkinson’s disease. Chlamydia pneumonia and trachoma also generate inclusion cysts and protein aggregates, which are found in Alzheimer’s disease, Parkinson’s disease, and other neurodegenerative diseases. Many chlamydia species…..
Today is World Alzheimer’s Day, and we take this opportunity to share a remarkable website, which has many experts in the field of intracellular pathogens serving its board. The group posts scientific articles supporting chlamydia pneumonia, trachoma, and emerging chlamydia species, as an infectious cause of Alzheimer’s, cardiovascular disease, asthma, arthritis, and multiple sclerosis. The website endorses a broader view of chronic disease, and provides educational information about chlamydia. The intracellular research group, and “The Origin of Disease: The War…..
Chronic infections cause Alzheimer’s. Findings in the brains of Alzheimer’s patients—plaque and tangles, abnormal proteins, and inflammation, are caused by pathogens and the response of the immune system. Chlamydia has been identified and cultured from Alzheimer’ brains, at the location of plaque and tangles, and inside microglia and immune cells. The microbiology of chronic chlamydia infection is consistent with the pathology found in Alzheimer’s brains. Retinal scans (OCTA) can view small blood vessels and measure neuronal loss, in the back…..
A NYT article reported scientists are finding mixed pathology in Alzheimer’s, including plaque, Lewy bodies, and silent stroke, which has made finding causes and cures significantly more difficult. The scientists are struggling to understand these findings and reframe the issues. Dr. Albert Hofman, chairman of the epidemiology department at Harvard, said he was convinced “Alzheimer’s disease is a vascular disease,” and the precipitating factor in Alzheimer’s is diminished blood flow to the brain. The article reported a more optimistic view…..
Medicine has now named a new form of dementia, called limbic-predominant age-related TDP-43 encephalopathy (LATE)—a type of dementia that mimics Alzheimer disease (AD) but is caused by TDP-43 protein deposits in the brain, rather than the beta-amyloid deposits of Alzheimer’s, and primarily affects people older than 80 years. The protein deposits in Parkinson’s, multiple sclerosis, and ALS are thought to be comprised of TDP-43. https://www.medscape.com/viewarticle/912556 More than one pathogen or combination of pathogens can cause the same disease; and an…..
No long-term study has been done to determine whether patients, who were previously treated with low-dose macrolides, cyclines, or quinolones, for acute and/or chronic infection, have a reduced risk of developing Alzheimer’s or Parkinson’s disease. The same immortal pathogens can cause cardiovascular disease; and studies showed patients treated in the prior three years with azithromycin, clarithromycin, or quinolones, for any reason, had a significant reduction in acute heart attack, stroke, and cardiac death. Male patients with cardiac disease and a…..
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