The cause of lupus has been hypothesized to be bacteria (Tb, chlamydia, H-pylori, streptococcus); viruses (EBV, Hep B & C, CMV, herpes 1/2/7); and/or genetics, the environment, trauma, or stress. One study compared 50 lupus patients with 50 normal patients, and found toxoplasmosis antibodies were significantly more common in lupus patients and lupus patients had particularly high titers of toxoplasmosis. Immortal pathogens and opportunistic infections trigger lupus and lupus exacerbations, by infecting immune cells; molecular mimicry; changes in apoptosis, generating nuclear and cell fragments; and changes in CD4 and CD 25 (regulators of T-cells), from abnormal proteins generated by the pathogen.
Chlamydia pneumonia and chlamydia trachoma have been repeatedly implicated in autoimmune disease and lupus, (as well as cardiovascular, neurologic, reproductive, cancer, and other chronic diseases). Chlamydia psittacosis has been repeatedly implicated in autoimmune disease, polyarthritis, psoriatic arthritis, psoriasis, melanoma, and cancer. When a patient has multiple types of immortal chronic infections, the pathogens generate a variety and abundance of abnormal proteins, which confuse weakened immune cells, generate inflammation, and impair organ function and flow.
Inflammatory arthritis and autoimmune disease are caused by chronic infections with immortal bacteria and parasites. Patients with autoimmune disease, inflammatory arthritis, and/or lupus, are entitled to diagnosis of pathogens and parasites capable of causing these diseases, alone or in combination. Finding the causes of inflammatory arthritis, autoimmune disease, and lupus; and helping patients with these diseases, begins with laboratory testing for immortal pathogens and parasites.
Wilcox M, et al. 1990. Toxoplasmosis and Systemic Lupus Erythemmatosus. Ann Rheum Dis. Apr 1990. 49: 254-257. Doi: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1004049/.
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