Chlamydia pathogens gain entry into the intestinal tract via sinus drainage and lung mucous; and find favorable host tissue along the intestinal tract. H-pylori is an immortal intestinal pathogen, which attacks epithelium, then burrows into and attaches to collagen, creating a portal through which other pathogens gain entry. Common parasitic infections begin as an intestinal infection (giardia, cryptosporidiosis, and/or worms), attach to the intestinal wall, block absorption of nutrition, and compete for nutrition.

Pathogens and parasites can migrate from the intestine to the pancreas, gallbladder, and liver, where the pathogens and parasites are protected from stomach acid. Pathogens and parasites in the pancreas, gallbladder, and liver provide a focus for immune system attack, stones, cysts, organ damage, and chronic disease.

Diabetic patients have chronic infections and are vulnerable to parasitic infection. They often develop a wide-belly shape, dysbiosis, metabolic syndrome, leaky gut syndrome, and fungus, from infectious pathogens and parasites. Diagnosing acute and chronic infections, in newly diagnosed and established diabetic patients, is the key to understanding causation, finding new treatments, and improving the patient’s quality of life.