We have numerous “biomes” within us—a microbiome (bacteria), mycobiome (fungus), proteinbiome (proteins), inflammasome (cells causing inflammatiom), and apoptosomes (cells affecting cell death). The gut-brain connection has been suggested in many chronic diseases, and science is studying “biomes” to better understand disease. All of the “biomes” are impacted by chronic infection and by treatments for infection. Chronic infection deposits and creates abnormal proteins, damages apoptosis, causes inflammation, and promotes the overgrowth of pathogenic fungus. Early in medical school, Dr. Merchant observed, during autopsies, that most deceased patients had disseminated fungus.
Penicillin changes the shape and extends the life expectancy of chlamydia species; broad-spectrum antibiotics kill both bad and good bacteria; and penicillin and broad-spectrum antibiotics present the greatest risk for antibiotic resistance. The problem of antibiotic resistance is not necessarily too many antibiotics being prescribed—it is too much penicillin and too many broad-spectrum antibiotics being prescribed; and animals consuming 80% of all antibiotics used, in the U.S. Tetracycline is even being fed to orange trees.
Rational antibiotic treatment must consider the pathogen; the adverse impact of untreated infection; and the adverse impact of treatment, on our “biomes”. The least expensive antibiotic is not necessarily the best antibiotic, in the short-term or long-term. The routine use of amoxicillin and broad-spectrum antibiotics, in humans and animals, and reluctance to prescribe any antibiotics due to fear of creating resistance, coincides with an increase in development of chronic diseases in younger patients and the development of antibiotic resistance. “Macrolides” and “cyclines” are more effective antibiotics against immortal pathogens, and are less likely to induce antibiotic resistance.
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